
Understanding Your Biofield: A Beginner's Guide
What is the human biofield, and how can measuring it reveal imbalances before they become symptoms? A deep dive into Gas Discharge Visualization and energy medicine.
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Karen sat in her doctor's office holding lab results. TSH: 2.8. "Normal," her doctor said. "Your thyroid is fine." But Karen didn't feel fine. Exhausted despite sleeping 9 hours. Cold all the time, wearing sweaters in summer. Weight climbing despite eating less than her husband. Hair thinning at the temples. Brain fog so thick she forgot words mid-sentence, lost her car in parking lots, re-read the same paragraph four times. "It must be stress," her doctor said. "Maybe depression. Have you tried exercising more?" Karen left feeling gaslit. Her labs said she was fine. Her body said she was falling apart. Here's the truth: TSH is a pituitary hormone. It measures your pituitary's signal to your thyroid. It does not measure thyroid hormone levels. It does not tell you how much active hormone is reaching your cells. You can have "normal" TSH and be profoundly hypothyroid at the tissue level. Karen's full thyroid panel, which her doctor never ordered, told a different story entirely.
TSH stands for Thyroid Stimulating Hormone. It's produced by your pituitary gland (in your brain) and signals your thyroid to produce thyroid hormone.
The feedback loop works like a thermostat. Your thyroid produces T4 and T3. Your pituitary senses thyroid hormone levels. If levels are low, the pituitary releases more TSH to tell the thyroid "make more." If levels are high, TSH drops.
TSH is an indirect measure. It tells you what your pituitary thinks is happening. Not what's actually happening in your cells.
Problems with relying on TSH alone:
TSH can be "normal" while Free T3 (the active hormone) is low. This happens when conversion from T4 to T3 is impaired.
Reverse T3 (the metabolic brake) isn't reflected in TSH at all. You could have sky-high Reverse T3 blocking every thyroid receptor in your body, and TSH won't budge.
Autoimmune thyroid disease (Hashimoto's) is invisible without antibody testing. Your thyroid can be under active autoimmune attack, and if the remaining tissue still produces enough hormone to satisfy the pituitary, TSH stays "normal."
Pituitary dysfunction from chronic stress, head trauma, or aging can cause inappropriately normal TSH despite low thyroid hormones.
Wadsworth and colleagues published a review in the Journal of Thyroid Research (2012) documenting cases of symptomatic hypothyroidism with normal TSH. It's not rare. It's under-recognized.
Your thyroid produces mostly T4, a storage hormone. T4 doesn't do much on its own. It must be converted to T3, the active hormone that drives metabolism, energy production, and every cellular process.
T4 is converted to T3 by enzymes called deiodinases in your liver, gut, and peripheral tissues. This conversion requires selenium, zinc, iron, iodine, a healthy gut, low inflammation, and low stress.
When conversion is impaired, T4 doesn't become T3. Instead, it's shunted into Reverse T3 (rT3), an inactive form that blocks thyroid receptors.
Free T3 is the metabolically active hormone. This is what you feel. Optimal range: upper third of the reference range, typically 3.0-4.0 pg/mL. When Free T3 is in the lower half of the range, you get fatigue, weight gain, cold intolerance, hair loss, brain fog, depression, constipation, and dry skin.
Reverse T3 is the metabolic brake. It sits on thyroid receptors and blocks T3 from binding. Optimal: below 15 ng/dL. Above 15 suggests conversion problems.
The critical ratio: (Free T3 x 100) / Reverse T3. Above 20 is optimal. Between 15-20 is suboptimal. Below 15 is cellular hypothyroidism.
Karen's full panel revealed: TSH 2.8 (normal). Free T4 mid-range (normal). Free T3 in the bottom 20% of the range (functionally low). Reverse T3: 24 ng/dL (elevated). Free T3/RT3 ratio: 12 (cellular hypothyroidism). TPO Antibodies: 180 (Hashimoto's, never tested before).
Her endocrinologist never ordered Free T3, Reverse T3, or antibodies. He saw TSH, said "normal," and sent her home.
Reverse T3 isn't inherently bad. It's a protective mechanism. Your body shifts T4 conversion toward Reverse T3 when it wants to slow metabolism.
Chronic stress: High cortisol impairs T4-to-T3 conversion and increases Reverse T3. This is your body conserving energy during perceived threat. Helmreich and Bhatt published the cortisol-Reverse T3 mechanism in Thyroid (2005).
Calorie restriction and dieting: When you under-eat, your body slows metabolism to conserve energy. T3 drops, Reverse T3 rises. This is why chronic dieting makes you cold, tired, and unable to lose weight. It's metabolic adaptation, not character failure.
Illness and inflammation: Acute or chronic illness shifts metabolism toward healing and away from high-energy activity. Reverse T3 rises during any inflammatory state. This is called "sick euthyroid syndrome" or "non-thyroidal illness."
Nutrient deficiencies: Selenium, zinc, and iron are required for proper conversion. Selenium deficiency is particularly relevant. Kohrle published extensively on selenium's role in thyroid hormone metabolism in Biochimie (1999).
Chronic infections: EBV, Lyme, mold, and other chronic infections drive inflammation and Reverse T3.
Liver dysfunction: The liver handles most T4-to-T3 conversion. Fatty liver, toxin burden, or liver inflammation all impair conversion.
Gut dysfunction: 20% of T4-to-T3 conversion happens in the gut via bacterial deiodinase activity. Dysbiosis, leaky gut, and gut inflammation reduce this conversion. Virili and Centanni reviewed this in European Review for Medical and Pharmacological Sciences (2017).
To lower Reverse T3, address the root causes. More thyroid medication without addressing why conversion is impaired just creates more substrate for Reverse T3 production.
Conventional medicine uses population-based reference ranges. These ranges are derived from the central 95% of a "reference population" that often includes people with undiagnosed thyroid disease. They catch severe disease. They miss dysfunction.
Functional ranges reflect optimal health, not just the absence of frank disease.
TSH: Conventional range is 0.5-5.0 mIU/L (some labs say 0.5-4.5). Functional optimal is 1.0-2.0 mIU/L. Wartofsky and Dickey argued for narrowing the TSH range to 0.4-2.5 in the Journal of Clinical Endocrinology & Metabolism (2005). TSH above 2.5 is associated with symptoms and increased cardiovascular risk.
Free T4: Optimal is upper-mid range (1.0-1.5 ng/dL). This is your storage hormone. Should be plentiful.
Free T3: Optimal is upper third of range (3.0-4.0 pg/mL). This is the active hormone. You want this high.
Reverse T3: Optimal is below 15 ng/dL. Above 15 suggests conversion problems.
Free T3/Reverse T3 ratio: Above 20 is optimal.
TPO Antibodies: Conventional "normal" is below 35 IU/mL. Functional optimal is below 9 IU/mL. Any elevation indicates autoimmune activity.
Thyroglobulin Antibodies: Conventional "normal" is below 40 IU/mL. Functional optimal is below 20 IU/mL. These antibodies are often elevated in Hashimoto's when TPO is normal. Test both.
The gap between "normal" and "optimal" is where millions of people live in misery, told they're fine when they're clearly not.
TSH alone misses most thyroid dysfunction. You need six tests:
This is the comprehensive thyroid assessment. Without all six, you're flying blind.
Most doctors won't order Free T3, Reverse T3, or antibodies. The reasons vary: insurance pushback, unfamiliarity with the markers, belief that TSH is sufficient, or guidelines that recommend against "unnecessary" testing.
You have options. Ask your doctor specifically for these tests. Explain that you have symptoms consistent with hypothyroidism despite normal TSH and want a complete picture. Some doctors will comply when patients advocate clearly.
If your doctor refuses, order them yourself through direct-to-consumer labs: Ulta Lab Tests, LabCorp, Quest Diagnostics. You pay out of pocket (typically $100-150 for the full panel) and get results without a doctor's order.
Don't accept "your thyroid is fine" based on TSH alone. That's like checking only the gas gauge when your car won't start. You need to check the battery, the starter, the spark plugs, and the fuel injectors too.
Levothyroxine (Synthroid, Levoxyl) is synthetic T4. It works well for many people who convert T4 to T3 efficiently.
But if you have conversion problems (stress, inflammation, nutrient deficiencies, gut issues), levothyroxine alone won't resolve symptoms. Your Free T3 will stay low while your TSH looks perfect.
Adding T3 (liothyronine): Synthetic T3 is dosed 2-3 times daily due to its short half-life. Typical dose: 5-25 mcg daily in divided doses. Many patients report dramatic improvement when T3 is added. Energy returns. Brain fog lifts. Weight loss becomes possible. Bunevicius and colleagues published a landmark study in NEJM (1999) showing that T4/T3 combination therapy improved mood and cognitive function compared to T4 alone.
Natural Desiccated Thyroid (NDT): Armour, Nature-Throid, NP Thyroid. Made from porcine thyroid glands. Contains T4, T3, T2, T1, and calcitonin: the full spectrum of thyroid hormones. NDT mimics human thyroid output more closely than synthetic T4 alone. Dose is measured in grains: 1 grain contains approximately 38 mcg T4 and 9 mcg T3.
Compounded T4/T3: Some patients do best with customized ratios (e.g., 100 mcg T4 + 10 mcg T3). Requires a compounding pharmacy.
The key: monitor Free T3 and symptoms, not just TSH. Optimize until Free T3 is in the upper third of the range and symptoms resolve. Some doctors will only dose by TSH, which means they'll reduce your medication the moment TSH drops below their comfort zone, even if you feel terrible. Find a doctor who treats the patient, not the number.
Adding thyroid hormone helps. But addressing why conversion is impaired is how you actually heal.
Optimize nutrients. Selenium (200 mcg daily) is required for conversion and reduces thyroid antibodies. Rayman published the evidence in Lancet (2012). Zinc (30 mg daily) is required for T4-to-T3 conversion. Iron (target ferritin above 70 ng/mL) is essential for TPO enzyme function. Iodine (150-300 mcg daily, cautiously) is required for thyroid hormone production. Vitamin D (optimize to 60-80 ng/mL) modulates immunity and thyroid function.
Reduce inflammation with omega-3s (2-3g EPA/DHA daily), curcumin (1-2g daily), and removal of inflammatory foods (gluten, seed oils, sugar).
Heal the gut. Gut dysfunction impairs conversion and nutrient absorption. Address SIBO, dysbiosis, leaky gut. Remove gluten, especially if you have Hashimoto's. Gliadin (gluten protein) cross-reacts with thyroid tissue through molecular mimicry.
Manage stress with adaptogenic herbs (ashwagandha, rhodiola, holy basil), meditation, breathwork, and sleep optimization.
Treat chronic infections. EBV reactivation, Lyme, mold: all impair thyroid function and drive Reverse T3.
Support liver detoxification. The liver converts most T4 to T3. Milk thistle, NAC, and glutathione support hepatic conversion capacity.
Karen implemented the full protocol. Gluten-free. Selenium, zinc, vitamin D. Ashwagandha for stress. Her doctor added 10 mcg liothyronine to her levothyroxine. Six months later: Free T3 in the upper third of the range. Reverse T3 down to 11. Ratio above 20. Energy restored. Hair growing back. Brain fog cleared. Weight dropping without dieting.
TSH is not thyroid function. It's one piece of a complex puzzle. You can have "normal" TSH and be profoundly hypothyroid. Low Free T3. High Reverse T3. Antibodies destroying your thyroid. Symptoms crushing your quality of life. Conventional medicine stops at TSH. Functional medicine investigates further. Demand comprehensive testing. Free T3, Reverse T3, antibodies. Calculate your Free T3/RT3 ratio. Interpret through functional ranges, not population averages built from sick people. If your doctor won't order these tests, find one who will. Or order them yourself. Your symptoms are real. Your labs, the right labs, will validate them. Optimize thyroid function, address root causes, and energy returns. You don't have to live in a fog. You don't have to accept being cold, tired, and foggy as your new normal. Your thyroid can work again. But first, you need to see the whole picture.

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